beta-Galactosidase Peptide Reference: GTX85284-PEP beta-Galactosidase peptide is used for blocking the activity of b-galactosidase antibody (catalog number GTX85284). It usually blocks the antibody activity completely in Western blot by incubating the peptide with equal volume of antibody for 3 min at 37oC.
Recombinant Human G-CSF Reference: GFH72-100 Granulocyte-Colony Stimulating Factor (G-CSF) is a cytokine that functions as a potent inducer of neutrophilic granulocyte proliferation, terminal differentiation, and activation. G-CSF synthesis occurs in monocyte, macrophage, epithelial, endothelial, and fibroblast cells after activation by bacterial endotoxins, Tumor Necrosis Factor α (TNF-α), Interleukin-1 (IL-1), or Interleukin-17 (IL-17). The functional activity of G-CSF is mediated through the granulocyte colony-stimulating factor receptor (G-CSF-R) to activate JAK/STAT and MAPK signal transduction pathways. G-CSF also promotes neurogenesis and inhibits neuronal apoptosis. Human and mouse G-CSF proteins are cross-reactive.
Siglec11 Peptide Reference: GTX85285-PEP Siglec11 peptide is used for blocking the activity of Siglec11 antibody (catalog number GTX85285). It usually blocks the antibody activity completely in Western blot by incubating the peptide with equal volume of antibody for 3 min at 37oC.
Recombinant Human G-CSF Reference: GFH72-1000 Granulocyte-Colony Stimulating Factor (G-CSF) is a cytokine that functions as a potent inducer of neutrophilic granulocyte proliferation, terminal differentiation, and activation. G-CSF synthesis occurs in monocyte, macrophage, epithelial, endothelial, and fibroblast cells after activation by bacterial endotoxins, Tumor Necrosis Factor α (TNF-α), Interleukin-1 (IL-1), or Interleukin-17 (IL-17). The functional activity of G-CSF is mediated through the granulocyte colony-stimulating factor receptor (G-CSF-R) to activate JAK/STAT and MAPK signal transduction pathways. G-CSF also promotes neurogenesis and inhibits neuronal apoptosis. Human and mouse G-CSF proteins are cross-reactive.
MATN3 Peptide Reference: GTX85286-PEP MATN3 peptide is used for blocking the activity of MATN3 antibody (catalog number GTX85286). It usually blocks the antibody activity completely in Western blot by incubating the peptide with equal volume of antibody for 3 min at 37oC.
Recombinant Human G-CSF (Animal-Free) Reference: GFH72AF-2 Granulocyte-Colony Stimulating Factor (G-CSF) is a cytokine that functions as a potent inducer of neutrophilic granulocyte proliferation, terminal differentiation, and activation. G-CSF synthesis occurs in monocyte, macrophage, epithelial, endothelial, and fibroblast cells after activation by bacterial endotoxins, Tumor Necrosis Factor α (TNF-α), Interleukin-1 (IL-1), or Interleukin-17 (IL-17). The functional activity of G-CSF is mediated through the granulocyte colony-stimulating factor receptor (G-CSF-R) to activate JAK/STAT and MAPK signal transduction pathways. G-CSF also promotes neurogenesis and inhibits neuronal apoptosis. Human and mouse G-CSF proteins are cross-reactive.
MATN1 Peptide Reference: GTX85287-PEP MATN1 peptide is used for blocking the activity of MATN1 antibody (catalog number GTX85287). It usually blocks the antibody activity completely in Western blot by incubating the peptide with equal volume of antibody for 3 min at 37oC.
Recombinant Human G-CSF (Animal-Free) Reference: GFH72AF-10 Granulocyte-Colony Stimulating Factor (G-CSF) is a cytokine that functions as a potent inducer of neutrophilic granulocyte proliferation, terminal differentiation, and activation. G-CSF synthesis occurs in monocyte, macrophage, epithelial, endothelial, and fibroblast cells after activation by bacterial endotoxins, Tumor Necrosis Factor α (TNF-α), Interleukin-1 (IL-1), or Interleukin-17 (IL-17). The functional activity of G-CSF is mediated through the granulocyte colony-stimulating factor receptor (G-CSF-R) to activate JAK/STAT and MAPK signal transduction pathways. G-CSF also promotes neurogenesis and inhibits neuronal apoptosis. Human and mouse G-CSF proteins are cross-reactive.
MATN3 Peptide Reference: GTX85288-PEP MATN3 peptide is used for blocking the activity of MATN3 antibody (catalog number GTX85288). It usually blocks the antibody activity completely in Western blot by incubating the peptide with equal volume of antibody for 3 min at 37oC.
Recombinant Human G-CSF (Animal-Free) Reference: GFH72AF-100 Granulocyte-Colony Stimulating Factor (G-CSF) is a cytokine that functions as a potent inducer of neutrophilic granulocyte proliferation, terminal differentiation, and activation. G-CSF synthesis occurs in monocyte, macrophage, epithelial, endothelial, and fibroblast cells after activation by bacterial endotoxins, Tumor Necrosis Factor α (TNF-α), Interleukin-1 (IL-1), or Interleukin-17 (IL-17). The functional activity of G-CSF is mediated through the granulocyte colony-stimulating factor receptor (G-CSF-R) to activate JAK/STAT and MAPK signal transduction pathways. G-CSF also promotes neurogenesis and inhibits neuronal apoptosis. Human and mouse G-CSF proteins are cross-reactive.
MATN1 Peptide Reference: GTX85289-PEP MATN1 peptide is used for blocking the activity of MATN1 antibody (catalog number GTX85289). It usually blocks the antibody activity completely in Western blot by incubating the peptide with equal volume of antibody for 3 min at 37oC.
Recombinant Human G-CSF (Animal-Free) Reference: GFH72AF-1000 Granulocyte-Colony Stimulating Factor (G-CSF) is a cytokine that functions as a potent inducer of neutrophilic granulocyte proliferation, terminal differentiation, and activation. G-CSF synthesis occurs in monocyte, macrophage, epithelial, endothelial, and fibroblast cells after activation by bacterial endotoxins, Tumor Necrosis Factor α (TNF-α), Interleukin-1 (IL-1), or Interleukin-17 (IL-17). The functional activity of G-CSF is mediated through the granulocyte colony-stimulating factor receptor (G-CSF-R) to activate JAK/STAT and MAPK signal transduction pathways. G-CSF also promotes neurogenesis and inhibits neuronal apoptosis. Human and mouse G-CSF proteins are cross-reactive.