Recombinant Human Islet cell autoantigen 1(ICA1),partial Reference: CSB-EP010947HU_100 May play a role in neurotransmitter secretion.
Recombinant Human Islet cell autoantigen 1(ICA1),partial Reference: CSB-EP010947HU_20 May play a role in neurotransmitter secretion.
Recombinant Human Islet cell autoantigen 1(ICA1) Reference: CSB-EP010947HU(F1)_1 May play a role in neurotransmitter secretion.
Recombinant Human Islet cell autoantigen 1(ICA1) Reference: CSB-EP010947HU(F1)_100 May play a role in neurotransmitter secretion.
Recombinant Human Islet cell autoantigen 1(ICA1) Reference: CSB-EP010947HU(F1)_20 May play a role in neurotransmitter secretion.
Recombinant Human Intercellular adhesion molecule 1(ICAM1),partial Reference: CSB-EP010949HU_1 ICAM proteins are ligands for the leukocyte adhesion protein LFA-1 (integrin alpha-L/beta-2). During leukocyte trans-endothelial migration, ICAM1 engagent promotes the assbly of endothelial apical cups through ARHGEF26/SGEF and RHOG activation. In case of rhinovirus infection acts as a cellular receptor for the virus.
Recombinant Human Intercellular adhesion molecule 1(ICAM1),partial Reference: CSB-EP010949HU_100 ICAM proteins are ligands for the leukocyte adhesion protein LFA-1 (integrin alpha-L/beta-2). During leukocyte trans-endothelial migration, ICAM1 engagent promotes the assbly of endothelial apical cups through ARHGEF26/SGEF and RHOG activation. In case of rhinovirus infection acts as a cellular receptor for the virus.
Recombinant Human Intercellular adhesion molecule 1(ICAM1),partial Reference: CSB-EP010949HU_20 ICAM proteins are ligands for the leukocyte adhesion protein LFA-1 (integrin alpha-L/beta-2). During leukocyte trans-endothelial migration, ICAM1 engagent promotes the assbly of endothelial apical cups through ARHGEF26/SGEF and RHOG activation. In case of rhinovirus infection acts as a cellular receptor for the virus.
Recombinant Human DNA-binding protein inhibitor ID-1(ID1) Reference: CSB-EP010966HU_1 Transcriptional regulator (lacking a basic DNA binding domain) which negatively regulates the basic helix-loop-helix (bHLH) transcription factors by forming heterodimers and inhibiting their DNA binding and transcriptional activity. Implicated in regulating a variety of cellular processes, including cellular growth, senescence, differentiation, apoptosis, angiogenesis, and neoplastic transformation. Inhibits skeletal muscle and cardiac myocyte differentiation. Regulates the circadian clock by repressing the transcriptional activator activity of the CLOCK-ARNTL/BMAL1 heterodimer .
Recombinant Human DNA-binding protein inhibitor ID-1(ID1) Reference: CSB-EP010966HU_100 Transcriptional regulator (lacking a basic DNA binding domain) which negatively regulates the basic helix-loop-helix (bHLH) transcription factors by forming heterodimers and inhibiting their DNA binding and transcriptional activity. Implicated in regulating a variety of cellular processes, including cellular growth, senescence, differentiation, apoptosis, angiogenesis, and neoplastic transformation. Inhibits skeletal muscle and cardiac myocyte differentiation. Regulates the circadian clock by repressing the transcriptional activator activity of the CLOCK-ARNTL/BMAL1 heterodimer .
Recombinant Human DNA-binding protein inhibitor ID-1(ID1) Reference: CSB-EP010966HU_20 Transcriptional regulator (lacking a basic DNA binding domain) which negatively regulates the basic helix-loop-helix (bHLH) transcription factors by forming heterodimers and inhibiting their DNA binding and transcriptional activity. Implicated in regulating a variety of cellular processes, including cellular growth, senescence, differentiation, apoptosis, angiogenesis, and neoplastic transformation. Inhibits skeletal muscle and cardiac myocyte differentiation. Regulates the circadian clock by repressing the transcriptional activator activity of the CLOCK-ARNTL/BMAL1 heterodimer .
Recombinant Human DNA-binding protein inhibitor ID-2(ID2) Reference: CSB-EP010967HU_1 Transcriptional regulator (lacking a basic DNA binding domain) which negatively regulates the basic helix-loop-helix (bHLH) transcription factors by forming heterodimers and inhibiting their DNA binding and transcriptional activity. Implicated in regulating a variety of cellular processes, including cellular growth, senescence, differentiation, apoptosis, angiogenesis, and neoplastic transformation. Inhibits skeletal muscle and cardiac myocyte differentiation. Regulates the circadian clock by repressing the transcriptional activator activity of the CLOCK-ARNTL/BMAL1 heterodimer. Restricts the CLOCK and ARNTL/BMAL1 localization to the cytoplasm. Plays a role in both the input and output pathways of the circadian clock: in the input component, is involved in modulating the magnitude of photic entrainment and in the output component, contributes to the regulation of a variety of liver clock-controlled genes involved in lipid metabolism.