Recombinant Rat Acetyl-CoA carboxylase 1(Acaca) ,partial Reference: CSB-EP001119RA_100 Catalyzes the rate-limiting reaction in the biogenesis of long-chain fatty acids. Carries out three functions: biotin carboxyl carrier protein, biotin carboxylase and carboxyltransferase.
Recombinant Rat Acetyl-CoA carboxylase 1(Acaca) ,partial Reference: CSB-EP001119RA_20 Catalyzes the rate-limiting reaction in the biogenesis of long-chain fatty acids. Carries out three functions: biotin carboxyl carrier protein, biotin carboxylase and carboxyltransferase.
Recombinant Human Medium-chain specific acyl-CoA dehydrogenase,... Reference: CSB-EP001126HU_1 This enzyme is specific for acyl chain lengths of 4 to 16.
Recombinant Human Medium-chain specific acyl-CoA dehydrogenase,... Reference: CSB-EP001126HU_100 This enzyme is specific for acyl chain lengths of 4 to 16.
Recombinant Human Medium-chain specific acyl-CoA dehydrogenase,... Reference: CSB-EP001126HU_20 This enzyme is specific for acyl chain lengths of 4 to 16.
Recombinant Human Acetyl-CoA acetyltransferase, mitochondrial(ACAT1) Reference: CSB-EP001134HU_200 Plays a major role in ketone body metabolism.
Recombinant Human Acetyl-CoA acetyltransferase, mitochondrial(ACAT1) Reference: CSB-EP001134HU_10 Plays a major role in ketone body metabolism.
Recombinant Human Acetyl-CoA acetyltransferase, mitochondrial(ACAT1) Reference: CSB-EP001134HUe1_1 Plays a major role in ketone body metabolism.
Recombinant Human Acetyl-CoA acetyltransferase, mitochondrial(ACAT1) Reference: CSB-EP001134HUe1_100 Plays a major role in ketone body metabolism.
Recombinant Human Acetyl-CoA acetyltransferase, mitochondrial(ACAT1) Reference: CSB-EP001134HUe1_20 Plays a major role in ketone body metabolism.
Recombinant Bovine Acetylcholinesterase(ACHE) Reference: CSB-EP001154BO_1 Terminates signal transduction at the neuromuscular junction by rapid hydrolysis of the acetylcholine released into the synaptic cleft.
Recombinant Bovine Acetylcholinesterase(ACHE) Reference: CSB-EP001154BO_100 Terminates signal transduction at the neuromuscular junction by rapid hydrolysis of the acetylcholine released into the synaptic cleft.